Skip Navigation

This Article
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by LANE, P. W.
Right arrow Articles by WOMACK, J. E.
Right arrow Search for Related Content
PubMed
Right arrow Articles by LANE, P. W.
Right arrow Articles by WOMACK, J. E.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

The Journal of Heredity 1979:70(2):133-135
© 1979 The American Genetic Association 70:133-135

other

Ashen, a new color mutation on chromosome 9 of the mouse

PRISCILLA W. LANE, and JAMES E. WOMACK

The authors are associated, respectively, with The Jackson Laboratory, Bar Harbor, Maine 04609 and The Institute of Comparative Medicine, Texas A & M University/Baylor College of Medicine, College Station, Texas 77843

Abstract

Ashen, a new recessive color mutation in the mouse, is described. It dilutes the coat color by affecting the shape of the melanocytes. The dendrites of ashen mice like those of leaden and dilute are smaller and thinner than those of non-ashen mice and the pigment granules are concentrated in the main body of the cell. Ashen has no significant effect on the activity levels of 11 liver lysosomal enzymes. Linkage tests show that it is on Chr 9 close to dilute and indicate that the most probable order of the four loci is Lap-I—14—Sep-I—22—ash—8—Mod-I.


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
 BloodHome page
E. K. Novak, R. Gautam, M. Reddington, L. M. Collinson, N. G. Copeland, N. A. Jenkins, M. P. McGarry, and R. T. Swank
The regulation of platelet-dense granules by Rab27a in the ashen mouse, a model of Hermansky-Pudlak and Griscelli syndromes, is granule-specific and dependent on genetic background
Blood, June 17, 2002; 100(1): 128 - 135.
[Abstract] [Full Text] [PDF]

Home page
 J. Cell Sci.Home page
X Wu, K Rao, M. Bowers, N. Copeland, N. Jenkins, and J. Hammer
Rab27a enables myosin Va-dependent melanosome capture by recruiting the myosin to the organelle
J. Cell Sci., January 3, 2001; 114(6): 1091 - 1100.
[Abstract] [PDF]

Home page
 Proc. Natl. Acad. Sci. USAHome page
S. M. Wilson, R. Yip, D. A. Swing, T. N. O'Sullivan, Y. Zhang, E. K. Novak, R. T. Swank, L. B. Russell, N. G. Copeland, and N. A. Jenkins
A mutation in Rab27a causes the vesicle transport defects observed in ashen mice
PNAS, July 5, 2000; 97(14): 7933 - 7938.
[Abstract] [Full Text] [PDF]


Disclaimer: Please note that abstracts for content published before 1996 were created through digital scanning and may therefore not exactly replicate the text of the original print issues. All efforts have been made to ensure accuracy, but the Publisher will not be held responsible for any remaining inaccuracies. If you require any further clarification, please contact our Customer Services Department.