Skip Navigation

This Article
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by Wallace, M. E.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Wallace, M. E.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

The Journal of Heredity 1985:76(4):271-278
© 1985 The American Genetic Association 76:271-278

research-article

An inherited agent of mutation with chromosome damage in wild mice

Margaret E. Wallace

The author is affiliated with the Department of Genetics, University of Cambridge, England. She thanks Professor S.C. Harland on whose farm the first interesting mice were obtained; Miss Olga Atteck made the first trapping and Dr. F. O'Gorman the second. Mr. J. Coppock, under Professor R. J. Berry's guidance, made the third. Successful breeding in the laboratory owes much to Misses J. M. Ferguson and E. J. Webster and Mrs. P. E. Fletcher. Mrs. S. R. Earnshaw gave valuable technical assistance. Dr. E. P. Evans and others gave advice on the cytological study, and Professor P. E. Polani's and Dr. Muriel Harris's discussion and advice is appreciated. The work was funded by the Medical Research Council.

Abstract

A population of wild mice in Peru was sampled three times over a period of 20 years. Inbreeding from each sample has shown the population to carry an unusually high frequency of visible mutants and nesting lethais. Preliminary evidence of chromosomal damage suggests and inherited tendency to some form of genetic instability.


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
 Proc. Natl. Acad. Sci. USAHome page
B. Wilm, E. Dahl, H. Peters, R. Balling, and K. Imai
Targeted disruption of Pax1 defines its null phenotype and proves haploinsufficiency
PNAS, July 21, 1998; 95(15): 8692 - 8697.
[Abstract] [Full Text] [PDF]

Home page
 DevelopmentHome page
P. Timmons, J Wallin, P. Rigby, and R Balling
Expression and function of Pax 1 during development of the pectoral girdle
Development, January 10, 1994; 120(10): 2773 - 2785.
[Abstract] [PDF]

Home page
 DevelopmentHome page
J Wallin, J Wilting, H Koseki, R Fritsch, B Christ, and R Balling
The role of Pax-1 in axial skeleton development
Development, January 5, 1994; 120(5): 1109 - 1121.
[Abstract] [PDF]

Home page
 DevelopmentHome page
H Koseki, J Wallin, J Wilting, Y Mizutani, A Kispert, C Ebensperger, B. Herrmann, B Christ, and R Balling
A role for Pax-1 as a mediator of notochordal signals during the dorsoventral specification of vertebrae
Development, January 11, 1993; 119(3): 649 - 660.
[Abstract] [PDF]


Disclaimer:
Please note that abstracts for content published before 1996 were created through digital scanning and may therefore not exactly replicate the text of the original print issues. All efforts have been made to ensure accuracy, but the Publisher will not be held responsible for any remaining inaccuracies. If you require any further clarification, please contact our Customer Services Department.