Skip Navigation

This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (4)
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by Novák, K.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Novák, K.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Journal of Heredity 2003:94(2)
© 2003 The American Genetic Association 94:191-193

Brief Communication

Allelic Relationships of Pea Nodulation Mutants

K. Novák

From the Institute of Microbiology, Academy of Sciences of the Czech Republic, Vídeská l083 Prague, CZ-142 20 Czech Republic.

Address correspondence to K. Novák at the address above, or e-mail: novak{at}biomed.cas.cz.

Thirteen stable nonnodulating mutant lines of pea (Pisum sativum L.) originating from cv. Finale were tested for allelism in pairwise crosses. The F1 plants were evaluated for the symbiotic phenotype under controlled growth conditions against the nodule bacterium Rhizobium leguminosarum bv. viciae strain 248. All mutations were found to be recessive and the lines were classified into eight complementation groups comprising Risnod1-Risnod23, Risnod8, Risnod9-Risnod22, Risnod14, Risnod19-Risnod25, Risnod20, Risnod24-Risnod26, and Risnod32. Position of Risnod21 was not firmly established, leaving the possibility of allelism both with Risnod19-Risnod25 and Risnod20. The results were partially consistent with the previous reports on the allelism of these lines. Additional crosses confirmed the correspondence of Risnod14 with the locus sym7 and of Risnod19-Risnod25 with sym8. The high number of eight complementation groups formed by 13 mutants provides an indication of additional nodulation loci in pea to those already reported and confirms the complexity of the genetic control of the early stages of nodulation.


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
 Crop Sci.Home page
K. Novak, M. Slajs, E. Biedermannova, and J. Vondrys
Development of an Asymbiotic Reference Line for Pea cv. Bohatyr by De Novo Mutagenesis
Crop Sci., August 1, 2005; 45(5): 1837 - 1843.
[Abstract] [Full Text] [PDF]

Home page
 Plant Physiol.Home page
P. Gepts, W. D. Beavis, E. C. Brummer, R. C. Shoemaker, H. T. Stalker, N. F. Weeden, and N. D. Young
Legumes as a Model Plant Family. Genomics for Food and Feed Report of the Cross-Legume Advances through Genomics Conference
Plant Physiology, April 1, 2005; 137(4): 1228 - 1235.
[Full Text] [PDF]


Disclaimer: Please note that abstracts for content published before 1996 were created through digital scanning and may therefore not exactly replicate the text of the original print issues. All efforts have been made to ensure accuracy, but the Publisher will not be held responsible for any remaining inaccuracies. If you require any further clarification, please contact our Customer Services Department.